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Saliva test for prostate cancer: How Accurate Is It?

By 3 January 2026January 18th, 2026No Comments

This piece explains why a new at‑home DNA risk tool has entered UK debate. BARCODE 1, published in the New England Journal of Medicine, assessed an at‑home spit sample that builds a polygenic risk score (PRS) to flag men at higher risk of prostate cancer. It is a genetic risk calculator rather than a simple yes/no diagnostic.

The process involves posting a small spit sample for DNA analysis. Results estimate future risk and help select who might need earlier screening.

Accuracy here means how well the PRS finds a high‑risk group in whom follow‑up screening uncovers cancers — not a replacement for MRI or biopsy. The headline figures show added value compared with PSA blood measurement for some men.

The article is an evidence‑led explainer that separates results from hype. Readers will learn how the PRS is built, what the trial shows, how PSA compares, and what NHS plans such as the TRANSFORM trial may mean for future screening.

Key Takeaways

  • BARCODE 1 used an at‑home spit route to produce a polygenic risk score.
  • The tool estimates genetic risk and does not diagnose disease directly.
  • Results suggest it may improve selection for further screening alongside PSA.
  • The evidence is preliminary; NHS evaluation continues via TRANSFORM.
  • Improved risk tools aim to identify men who should be investigated sooner.

Why the saliva ‘spit test’ is making headlines in the UK

A UK research collaboration has put a home-collected DNA risk measure in the spotlight. Researchers at the Institute of Cancer Research, London, and the Royal Marsden NHS Foundation Trust reported BARCODE 1 results showing a polygenic risk score derived from a postal sample can better identify some men at higher future risk than the PSA blood check.

What the Institute of Cancer Research and Royal Marsden NHS teams announced

The teams said the approach can be done at home and still produce useful genetic risk information. This drew media attention because it could help target clinic resources and reduce unnecessary appointments.

Why screening needs tools beyond the PSA test

PSA remains part of GP pathways: risk assessment, GP testing and onward referral for imaging and biopsy. But PSA has limits — many raised results are false positives and some cancers occur despite a normal PSA reading.

That combination of false alarms and missed cases explains why the new tool is seen as a potential addition rather than a direct replacement. For patients and GPs, a better risk-based strategy could mean fewer unnecessary scans and earlier diagnosis of those at higher baseline risk.

  • Practical point: the new approach aims to refine who should be invited for further screening.
  • Policy note: there is no national screening programme in the UK, so targeted tools are of active interest.

What the BARCODE 1 study tested and who took part

BARCODE 1 used a mailed home kit so participants could provide a small spit sample, making DNA collection simple and scalable.

At-home sample collection and DNA extraction

Men returned the kits by post. Labs extracted DNA and ran genetic analysis to produce a combined risk score.

The men included

The study analysed material from 6,142 European-heritage men aged 55–69, recruited through GP surgeries. This age group was chosen because risk rises and screening impact is greater.

How polygenic risk scores are built

A polygenic risk score (PRS) adds together the tiny effects of many inherited changes. BARCODE 1 used 130 genetic variants linked prostate cancer, based on datasets of hundreds of thousands of men.

Key point: the PRS estimates genetic risk and the trial then checked whether targeting high-risk men raised cancer detection on follow-up. A limitation is that the research focused on men of European ancestry, so broader validation is needed.

Item Value Notes
Participants 6,142 men Age 55–69; recruited via GP surgeries
Sample type Spit (mailed) Home collection enabled scale
Variants used 130 Derived from large genetic studies
Score purpose Estimate genetic risk Used to target high-risk men for follow-up

Saliva test for prostate cancer accuracy: what the results show

BARCODE 1 used a risk-first pathway that moved only the men at highest genetic risk into imaging and biopsy. This approach measured how well inherited scores flag people who actually have disease on clinical follow-up.

High-risk identification and invitations

The study invited the top 10% by polygenic scores to undergo MRI and biopsy. By concentrating resources on a small group, the trial tested whether genetic selection can better identify men highest risk than broad screening.

Detection after MRI and biopsy

After targeted MRI and biopsy, around 40% of those invited were diagnosed — 187 men in the reports. That is a high detection yield compared with typical population screening.

How many cancers PSA could miss

A striking finding was the number with a normal PSA level. Sources report roughly 63.1% (118/187) in one analysis and 77.8% (147/187) in another had PSA below 3.0 µg/L.

This suggests PSA alone can miss a large share of tumours in men with high genetic risk, supporting the idea that a genetic risk filter helps identify men who need further assessment.

Aggressive disease detection

Importantly, PRS-identified cancers included a higher proportion of clinically significant or aggressive cancers (55.1%) versus 35.5% in a recent PSA-based study. The score therefore picked up more serious disease, not only low-grade lesions.

Important nuance: the genetic score does not diagnose aggressive cancers by itself; it prioritises who should receive definitive tests such as an MRI scan and biopsy.

How it compares with the PSA blood test

Clinicians compare the familiar PSA blood check with a genetic risk score to see which better guides follow-up care.

False positives and overdiagnosis

PSA can give false alarms. Around three out of four raised results do not indicate a dangerous condition. That leads to many men being offered an MRI scan and sometimes an invasive biopsy.

Overdiagnosis is also a concern. PSA can detect slow-growing tumours that may never cause symptoms, creating pressure to treat when monitoring might suffice.

PSA thresholds and what “raised” means

In UK reports a common reference level is 3.0 µg/L. Above that, clinicians often discuss onward imaging or referral.

That threshold influences who enters further screening and who remains under routine care.

Where the PRS approach adds value

The polygenic route acts as a stratification tool. BARCODE 1 suggests it may flag men higher risk who a PSA check alone might miss.

“Using inherited-risk information can focus scarce NHS resources on those most likely to benefit.”

Measure Typical outcome Practical impact
PSA blood check Many false positives (≈75%) More MRI scans and biopsies
PRS genetic score Targets men higher risk May reduce unnecessary scans
Combined approach Higher yield of clinically significant disease Smarter screening and resource use

More accurate than an MRI scan for some high genetic-risk men

In men flagged as high genetic risk, BARCODE 1 found that MRI did not spot a large share of biopsy-confirmed disease. This surprising result shows imaging has limits when used in a targeted pathway.

Biopsy-confirmed cancers that MRI did not detect

Key statistic: roughly two-thirds of biopsy-proven prostate cancer cases in the high-risk group were not seen on MRI—reported as 66.8% (125 men) and 63.6% (119 men) in different analyses.

What “MRI missed” means: the scan showed no suspicious lesion, yet biopsy found cancer. MRI therefore cannot be treated as a definitive rule-out in these men.

  • The finding emphasises that genetic selection and imaging serve different roles.
  • A combined pathway can direct who needs further assessment even after a negative mri scan.
  • Relying on MRI alone in high-risk men could leave many cancers undetected.
Measure Result Implication
High-risk group Top decile by genetic score Targeted imaging and biopsy
Missed by MRI ≈64–67% of biopsy cases Scan not definitive
Practical point Combined approach PRS helps decide who should have biopsy despite a negative mri

Who the test may help most and what it means for risk-based screening

New genetic tools seek to distinguish which men need earlier investigation from those who can be safely monitored. The aim is to concentrate NHS resources on men most likely to develop clinically important disease.

Targeting age, ethnicity and inherited risk

Discussion in the UK centres on age (mid‑50s onward), ethnic background and inherited genetic risk. These factors combine to help identify men who may benefit from more frequent checks or earlier imaging.

Extending to diverse ancestry groups

BARCODE 1 focused on men of European heritage. The Institute of Cancer Research has since developed PRODICT® to perform better in Asian and African ancestry groups. This work matters because Black men are about twice as likely to be diagnosed with prostate cancer.

Related studies shaping evidence

PROFILE and other projects are examining risk markers in Black men and those with a family history. Ongoing cancer research will help refine which men higher risk need targeted follow-up.

What a national programme could look like

A future UK screening pathway might start with a simple risk assessment that includes genetics, then offer tailored screening intervals and focused use of MRI and biopsy. These studies are guiding that direction, but routine access to a spit test is not yet standard NHS practice.

What happens next: TRANSFORM trial, NHS impact and timelines

The £42m TRANSFORM trial will compare inherited-risk screening directly with current NHS approaches. It is funded and instigated by Prostate Cancer UK and partly led by the Institute of Cancer Research team. The trial will run head-to-head, assessing genetic risk via a mailed kit, PSA blood checking and MRI pathways.

How the trial will compare approaches

The design tests which route, or combination, best finds clinically important disease and reduces unnecessary procedures. TRANSFORM will measure detection rates, false positives and downstream resource needs.

Potential benefits and projected NHS impact

Researchers estimate the programme could identify up to 12,350 people earlier and save the NHS around £500m a year. These are projections, not guaranteed outcomes, but they frame why the trial is decisive.

Expert views, practical steps and timelines

Prostate Cancer UK stresses that the PSA blood check remains the best available, accessible option via GPs today. If TRANSFORM shows clear gains, national rollout would need peer review, guideline updates and expanded MRI and biopsy capacity.

“TRANSFORM is designed to establish the most accurate and cost-effective screening pathway for the NHS.”

Aspect TRANSFORM aim Projected figure
Earlier identification Find clinically significant disease sooner Up to 12,350 people
Estimated savings Reduce unnecessary procedures and costs ≈£500m per year
Policy step Evidence to inform NHS roll-out Depends on results, review and planning

Conclusion

The trial indicates inherited-risk screening may sharpen who receives further assessment. In short, the BARCODE 1 approach looks promising as a risk tool, not a standalone diagnosis.

The study showed a high detection yield — about 40% in the top genetic-risk group — and many tumours occurred at normal PSA levels. MRI also missed a notable share of biopsy-confirmed cases, underlining limits of imaging alone.

If larger trials confirm benefit, screening could shift to fewer unnecessary investigations and earlier, focused diagnosis for men at higher risk. For now, PSA remains the routine NHS test and postal saliva kits are still under evaluation by the Institute Cancer Research and partners.

Men with concerns should discuss personal risk and next steps with their GP.

FAQ

What did researchers at the Institute of Cancer Research and The Royal Marsden NHS Foundation Trust announce?

They reported a non-blood spit-based genetic screening approach that uses DNA to estimate inherited risk. The study showed the approach can identify men at higher lifetime risk and direct those at greatest risk to further scans and biopsy assessments.

How was the BARCODE 1 study set up and who took part?

BARCODE 1 enrolled more than 6,000 men aged 55–69 via GP surgeries. Participants sent a home-collected saliva sample for DNA analysis, and those in the top genetic-risk decile were invited for MRI and biopsy if indicated.

How are polygenic risk scores (PRS) generated in this work?

PRS combine information from around 130 inherited genetic markers associated with disease risk. Each variant adds a small amount to overall risk; together they stratify men into higher or lower genetic-risk groups.

What did the results show about identifying men at highest risk?

The top 10% by PRS were invited for further screening. This targeted approach increased the chance of finding cancer among men offered MRI and biopsy compared with an unselected screening strategy.

How many cancers were detected after MRI and biopsy in the high-risk group?

About four in ten men who underwent MRI and biopsy following high genetic-risk selection were diagnosed with prostate malignancy, including a substantial proportion of clinically significant disease.

Can men have a normal PSA level yet still have cancer detected through this pathway?

Yes. The study found cases where men had PSA levels considered normal but were found to have cancer after imaging and biopsy, showing inherited risk information can reveal cancers that PSA alone might miss.

How does this genetic approach compare with the PSA blood test?

The PRS-based route seeks to reduce unnecessary imaging and biopsies by targeting men at higher inherited risk. PSA remains useful but can produce false positives and lead to overdiagnosis; combining genetic risk with PSA and MRI may improve precision.

Does the polygenic approach reduce false positives and overdiagnosis seen with PSA screening?

It can reduce unneeded procedures by narrowing who is offered MRI and biopsy. By focusing on men with elevated inherited risk, clinicians can limit scans for those unlikely to benefit while still finding more clinically important tumours in the targeted group.

Were there cancers confirmed by biopsy that MRI missed in the study?

Yes. Some biopsy-proven tumours were not visible on MRI, indicating the genetic-risk selection can identify men who may harbour disease even when imaging appears normal.

Who stands to benefit most from a risk-based screening model?

Men with higher inherited risk, older age within the studied cohort, and those from ethnic groups with elevated incidence could gain most. Targeting by genetic risk aims to catch aggressive disease earlier in those most likely to develop it.

How is the approach being extended to diverse populations?

Follow-up studies such as PRODICT® and international research efforts aim to validate and adapt risk scores for men of Black, Asian and African ancestry, ensuring equitable accuracy across different genetic backgrounds.

What related studies are helping shape the evidence base?

Trials including PROFILE and other observational studies complement BARCODE 1 by testing targeted screening, refining risk algorithms and assessing outcomes from combining inherited risk with conventional tools.

What will the TRANSFORM trial evaluate next?

TRANSFORM will compare a genetic-risk based pathway directly with standard PSA-led approaches and with MRI strategies. The trial will assess detection rates, stage and grade of cancers found, and health-economic impacts for the NHS.

What are the potential benefits for the NHS if this pathway is adopted?

Potential advantages include earlier detection of clinically significant disease, fewer unnecessary scans and biopsies, and long-term cost savings if targeted screening prevents advanced cases requiring intensive treatment.

Why do experts still consider PSA the current best available test?

PSA remains the established, widely available blood marker supported by decades of evidence and clinical pathways. Researchers emphasise that any new screening approach must be validated in randomised trials before replacing PSA in routine practice.

Could a national screening programme in the UK use genetic-risk stratification?

In future, a national programme might combine inherited risk scores with PSA and MRI to target men most likely to benefit. Policymakers will await results from ongoing trials, cost-effectiveness analyses and equity assessments before making decisions.